Coconut Oil has naturally occuring anti viral properties. Best known are its fatty acids. There are numerous scholarly publications with regards to fatty acids & their effect on lipid-enveloped viruses. Coconut Oil is a natural source of these fatty acids.
Monolaurin alone and monolaurin with tert-butylhydroxyanisole (BHA), methylparaben, or sorbic acid were tested for in vitro virucidal activity against 14 human RNA and DNA enveloped viruses in cell culture. At concentrations of 1% additive in the reaction mixture for 1 h at 23°C, all viruses were reduced in infectivity by >99.9%. Monolaurin with BHA was the most effective virucidal agent in that it removed all measurable infectivity from all of the viruses tested. The compounds acted similarly on all the viruses and reduced infectivity by disintegrating the virus envelope. All rights reserved.
The enveloped bacteriophage φ6 has been shown to be an interesting model system for the study of chemical agents that might have specific antiviral effects against lipid-containing mammalian viruses. In this report, we describe two types of antiviral activity exhibited by several fatty acids against bacteriophage φ6. Oleic acid (18:1) and palmitoleic acid (16:1) were potent inactivators of the virus. Treatment with either fatty acid at 50 ,ug/ml at 25 or 0°C for 30 min reduced the virus titer to about 0.1% ofthe initial titer. Oleic acid at a concentration as low as 3 ,ug/ml (.10-2 mM) reduced the virus titer to <1% of the initial titer within 30 min. Ultracentrifugation analyses of 14C-amino acid- and 32P labeled virus treated with oleic acid indicated that the virion is largely disassemble'd by the treatment. Myristic acid (14:0) and palmitic acid (16:0) did not inactivate φ6 at 50 ,ug/ml, but nevertheless did prevent φ6 plaque production. Single-step virus growth experiments in which fatty acid was added at various times before or after infection indicated that it was an early stage of the φ6 replication cycle that was inhibited by the presence of myristic acid and that the inhibition occurred only if the myristic acid concentration in the extracellular growth medium was 10 ,ug/ml. φ6 could attach to its host cell in the presence of myristic acid at 50 ,ug/ml. We conclude that the fatty acids that prevent φ6 replication probably do so by interfering with the entry of the viral genome into the host cell. This report provides the first indication that fatty acids can inactivate and inhibit the replication of a lipid-containing virus. Since fatty acids do not have toxic effects on some cells at the concentrations used here (but see reference 14), it appears that fatty acids should be tested further, using several lipid-containing mammalian viruses, to investigate the possible clinical use of fatty acids as inhibitory agents against certain viruses. All rights reserved.
Wallace Snipes,* Stanley Person, Gregory Keller, William Taylor, and Alec Keith, Biophysics Laboratory, Department of Biochemistry and Biophysics, The Pennsylvania State University, University Park, Pennsylvania 16802, Received for publication 9 July 1976. This report describes the inactivation of lipid-containing viruses by several long-chain alcohols. A striking peak in antiviral activity was found for saturated alcohols having chain lengths from 10 to 14 carbons. Viruses having different membrane structure showed different susceptibilities to alcohols having different chain lengths and structural features. Decanol, dodecanol, and tetradecanol readily inactivated herpes simplex virus and the enveloped bacterial virus φ6. The lipid-containing virus PM2 was susceptible to decanol and dodecanol but comparatively unsusceptible to tetradecanol. The branched-chain alcohol phytol, a naturally occurring component of chlorophyll, was active against φ6 and herpes simplex virus but not against PM2. Polyoma virus and the bacteriophage 423-1-a, which do not contain lipids, were not susceptible to inactivation by any of the alcohols tested. Experiments were also carried out to determine the effects of these compounds on cells. At 0.5 mM, decanol lysed human embryonic lung cells, erythrocytes, and the bacterial hosts for φ6 and PM2. Dodecanol, tetradecanol, and phytol at this concentration were less damaging to cells. At 0.05 mM, none of the alcohols caused observable cytopathic effects on human embryonic lung cells, although several of the alcohols at this concentration were active against herpes simplex virus. Our findings suggest that dodecanol, tetradecanol, and phytol may warrant further studies as potential antiviral agents, particularly for topical application to virus-infected areas of the skin. All rights reserved.
Coconut oil is a fat consisting of about 90% saturated fat. The oil contains predominantly medium chain triglycerides, with roughly 92% saturated fatty acids, 6% monounsaturated fatty acids, and 2% polyunsaturated fatty acids. Of the saturated fatty acids, coconut oil is primarily 44.6% lauric acid, 16.8% myristic acid , 8.2% palmitic acid and 8% caprylic acid. Although it contains seven different saturated fatty acids in total, its only monounsaturated fatty acid is oleic acid while its only polyunsaturated fatty acid is linoleic acid. 1 Nutrition Facts and Information for Vegetable oil, coconut 2 Nutrient analysis of coconut oil – USDA All rights reserved
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